SCCM Resource Library-Neuroprotective Effects of Pharmacologic Agent JTE-013 After Cardiac Arrest in Rats

Neuroprotective Effects of Pharmacologic Agent JTE-013 After Cardiac Arrest in Rats

Video Transcription

Video Summary

In this video, the speaker discusses a study that explores the effects of a potential therapeutic intervention called JTE013 on neural inflammation and ischemic brain injury induced by cardiac arrest. The study involves both in vitro and in vivo models. In the in vitro model, JTE013 is shown to protect neurons, myelin, and synapses. In the in vivo model, rats treated with JTE013 after cardiac arrest have improved neurological outcomes compared to control rats. The results suggest that JTE013 could be a promising neuroprotective treatment for brain injury caused by cardiac arrest. The study is supported by NIH funding.

Asset Subtitle

Cardiovascular, 2023

Asset Caption

Type: star research | Star Research Presentations: Neuroscience (SessionID 30005)

Meta Tag

Content Type Presentation

Knowledge Area Cardiovascular

Membership Level Professional

Membership Level Select

Tag Cardiac Arrest

Year 2023

Keywords

neuroinflammation

ischemic brain injury

JTE013

neuroprotective treatment

cardiac arrest

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